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why does my lower stomach keep twitching

Identifying the cause of your stomach spasms can help you treat this symptom. Here are 11 conditions that may be responsible for your symptom. 1. Muscle strain
Overworking your abdominal muscles could cause them to spasm. Spasms due to muscle strain are most likely to occur in people who do strenuous and frequent exercise, especially crunches and situps. 2. Dehydration Losing electrolytes from caused by sweating, vomiting, and diarrhea can result in muscle spasms throughout your body, including your stomach. This happens because muscles need electrolytes such as calcium, and to work properly. When they donБt have these electrolytes, your muscles may start working abnormally and seizing up. 3. Gas A buildup of gas in your stomach can cause your intestinal muscles to spasm as your body tries to release the gas. If you have gas, you might also have: 4. Inflammatory bowel disease These diseases, such as and, are chronic inflammatory conditions. CrohnБs disease can affect any part of the gastrointestinal tract, while UC only affects the colon. In both conditions, inflammation can cause bowel spasms. 5. Irritable bowel syndrome is a chronic condition that affects the large intestine. It doesnБt cause bowel tissue changes like, but symptoms are similar, including: 6.

Gastritis and gastroenteritis and are both stomach inflammation, but in gastroenteritis, the intestines are also inflamed. Infections, such as from Helicobacter pylori, Norwalk virus, and rotavirus, usually cause these conditions. 7. Infectious colitis Colitis can cause abdominal cramping due to irritation and inflammation of the colon, which causes it to spasm. Some bacteria that can cause colitis include Clostridium, Salmonella, and E. coli. Parasites such as Giardia can cause colitis too. 8. Ischemic enteritis and colitis Sometimes colitis is caused by lack of blood supply to the small bowel and colon. Spasms can occur in this type of colitis as well. 9. Constipation Your bowels may cramp when you experience constipation as they distend in response to increased pressure inside them. 10. Ileus An ileus is when your bowels become БlazyБ or Бsleepy. Б This can occur for a number of reasons including infection, inflammation, recent surgery (especially in the abdomen), narcotic use, severe illness, and lack of physical activity. An ileus causes your bowels to fill with air and fluid, resulting in distention and pain. 11. Gastroparesis is basically an ileus involving the stomach. It most commonly occurs in those with and can cause stomach cramping especially after eating.

Celiac. com 05/16/2018 - Galectins are a family of animal lectins marked by their affinity for N-acetyllactosamine-enriched glycoconjugates. Galectins control several immune cell processes and influence both innate and adaptive immune responses. A team of researchers recently set out to assess the role of galectins, particularly galectin-1 (Gal-1), in the treatment of celiac disease. The research team included Victoria Sundblad, Amado A. Quintar, Luciano G. Morosi, Sonia I. Niveloni, Ana Cabanne, Edgardo Smecuol, Eduardo Maurio, Karina V. Mario, Julio C. Bai, Cristina A. Maldonado, and Gabriel A. Rabinovich. The researchersPexamined the role of galectins in intestinal inflammation, particularly in Crohns disease, ulcerative colitis, and celiac disease patients, as well as in murine models resembling these inflammatory conditions. P Maintaining the fine balance between host immunity and tolerance promotes gut homeostasis, and helps to prevent inflammation. To gain insight into the role of Gal-1 in celiac patients, the team demonstrated an increase in Gal-1 expression following a gluten-free diet along with an increase in the frequency of Foxp3+ cells. P The resolution of the inflammatory response may promote the recovery process, leading to a reversal of gut damage and a regeneration of villi.

Among other things, the teams findings support the use of Gal-1 agonists to treat severe mucosal inflammation. In addition, Gal-1 may serve as a potential biomarker to follow the progression of celiac disease treatment. Gut inflammation may be governed by a coordinated network of galectins and their glycosylated ligands, triggering either anti-inflammatory or pro-inflammatory responses. PThat network may influence the interplay between intestinal epithelial cells and the highly specialized gut immune system in physiologic and pathologic settings. The teams results demonstrate that the anti-inflammatory and tolerogenic response associated with gluten-free diet in celiac patients is matched by a substantial up-regulation of Gal-1. This suggests a major role of this lectin in favoring resolution of inflammation and restoration of mucosal homeostasis. P This data highlights the regulated expression of galectin-1 (Gal-1), a proto-type member of the galectin family, during intestinal inflammation in untreated and treated celiac patients. Further study of this area could lead to better understanding of the mechanisms behind celiac disease, and potentially to a treatment of the disease.

Front. Immunol. , 01 March 2018. P The researchers in this study arePvariously affiliated with the Laboratorio de Inmunopatologa, Instituto de Biologa y Medicina Experimental (IBYME), Consejo Nacional de Investigaciones Cientficas y Tcnicas (CONICET), Buenos Aires, Argentina; the Centro de Microscopa Electrnica, Facultad de Ciencias Mdicas, Universidad Nacional de Crdoba, Crdoba, Argentina; the Instituto de Investigaciones en Ciencias de la Salud (INICSA), Consejo Nacional de Investigaciones Cientficas y Tcnicas (CONICET), Crdoba, Argentina; the Laboratorio de Glicmica Funcional y Molecular, Instituto de Biologa y Medicina Experimental (IBYME), Consejo de Investigaciones Cientficas y Tcnicas (CONICET), Buenos Aires, Argentina; the Seccin Intestino Delgado, Departamento de Medicina, Hospital de Gastroenterologa Dr. C. Bonorino Udaondo, Buenos Aires, Argentina; the Unidad de Patologa, Hospital de Gastroenterologa, Bonorino Udaondo, Buenos Aires, Argentina; the Instituto de Investigaciones, Universidad del Salvador, Buenos Aires, Argentina; and the Departamento de Qumica Biolgica, Facultad de Ciencias Exactas y Naturales, Universidad de Buenos Aires, Buenos Aires, Argentina.

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