why does my body smell like fish

*Fish odor syndrome (trimethylaminuria) facts Medically Edited by:
Charles P. Davis, MD, PhD Fish odor syndrome (trimethylaminuria) is a ; symptoms are often present from birth. Fish odor syndrome is characterized by an offensive body odor and the smell of rotting fish due to the excessive excretion of trimethylaminuria (TMA) in the urine, sweat, and breath of affected individuals. Fish odor syndrome is caused by mutations in the FMO3 gene. Other causes of odor body can occur as a result from an excess of proteins in the, or from an increase in bacteria that normally produce trimethylamine in the GI tract (also may make fish odor syndrome worse); disease, and cases of blood-borne. Diagnostic tests that help differentiate fish odor syndrome (trimethylaminuria) from other causes of body odor are available at specialized laboratories.

Fish odor syndrome symptoms have been seen temporarily in a small number of premature infants and in healthy women at the start of, usually in individuals that are heterozygous for the mutant FMO3 gene or due to immaturity of the faulty FMO3 gene expression ( FMO3 expression increases with age). Fish odor syndrome treatment includes certain dietary restrictions, use of acid lotions and soaps to remove secreted trimethylamine on the skin, use of activated charcoal and copper chlorophyllin, certain antibiotics, laxatives, and riboflavin. Mitchell SC, Smith RL (2001). "Trimethylaminuria: the fish malodor syndrome". Drug Metab Dispos. 29 (4 Pt 2): 51721. P. Treacy EP, et al. (1998). "Mutations of the flavin-containing monooxygenase gene ( FMO3 ) cause trimethylaminuria, a defect in detoxication".

Human Molecular Genetics. 7 (5): 83945. :. P. Zschocke J, Kohlmueller D, Quak E, Meissner T, Hoffmann GF, Mayatepek E (1999). "Mild trimethylaminuria caused by common variants in FMO3 gene". Lancet. 354 (9181): 8345. :. P. Mackay RJ, McEntyre CJ, Henderson C, Lever M, George PM (2011). Clin Biochem Rev. 32 (1): 3343. P. CS1 maint: Multiple names: authors list ( ^. Retrieved. Hernandez D, Addou S, Lee D, Orengo C, Shephard EA, Phillips IR (2003). "Trimethylaminuria and a human FMO3 mutation database". Hum Mutat. 22 (3): 20913. :. P. Furnes B, Feng J, Sommer SS, Schlenk D (2003). "Identification of novel variants of the flavin-containing monooxygenase gene family in African Americans".

Drug Metab Dispos. 31 (2): 18793. :. P. Allerston CK, Shimizu M, Fujieda M, Shephard EA, Yamazaki H, Phillips IR (2007). "Molecular evolution and balancing selection in the flavin-containing monooxygenase 3 gene (FMO3)". Pharmacogenet Genomics. 17 (10): 82739. :. P. ^ Shephard, Elizabeth A; Treacy, Eileen P; Phillips, Ian R (30 November 2011). European Journal of Human Genetics. 20 (3). :. Lenherr N, Berndt A, Ritz N, Rudin C. Aerococcus urinae: a possible reason for malodorous urine in otherwise healthy children. Eur J Pediatr. 2014;173:1115-7; Gibb AP, Sivaraman B. A second case of foul smelling urine in a boy caused by Aerococcus urinae. Pediatr Infect Dis J. 2013;32:1300-1. ^ Mountain H, Brisbane JM, Hooper AJ, Burnett JR, Goldblatt J (2008). "Trimethylaminuria (fish malodour syndrome): a "benign" genetic condition with major psychosocial sequelae".

Med. J. Aust. 189 (8): 468. P. Treacy E; Johnson D. Pitt JJ; Danks DM (1995). "Trimethylaminuria, fish odour syndrome: A new method of detection and response to treatment with metronidazole". J Inherit Metab Dis. 18 (3): 30612. :. P. Yamazaki H, Fujieda M, Togashi M, et al. (2004). "Effects of the dietary supplements, activated charcoal and copper chlorophyllin, on urinary excretion of trimethylamine in Japanese trimethylaminuria patients". Life Sci. 74 (22): 273947. :. P. Humbert JA, Hammond KB, Hathaway WE (1970). "Trimethylaminuria: the fish-odour syndrome". Lancet. 2 (7676): 7701. :. P.

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